Instabilotyping reveals unique mutational spectra in microsatellite-unstable gastric cancers.

نویسندگان

  • Yuriko Mori
  • Fumiaki Sato
  • Florin M Selaru
  • Andreea Olaru
  • Kellie Perry
  • Martha C Kimos
  • Gen Tamura
  • Nagahide Matsubara
  • Suna Wang
  • Yan Xu
  • Jing Yin
  • Tong-Tong Zou
  • Barbara Leggett
  • Joanne Young
  • Toshihiro Nukiwa
  • O Colin Stine
  • John M Abraham
  • David Shibata
  • Stephen J Meltzer
چکیده

Microsatellite instability (MSI) within coding regions causes frameshift mutations (FSMs). This type of mutation may inactivate tumor suppressor genes in cancers with frequent MSI (MSI-H cancers). To identify novel FSMs in gastric carcinogenesis in an unbiased and comprehensive manner, we screened for this type of mutation at 154 coding region repeat loci in 18 MSI-H gastric cancers. We also compared FSM rates and spectra in MSI-H gastric versus colorectal cancers. Thirteen novel loci showed FSMs in >20% of gastric tumors. Novel loci with the highest mutation frequencies included the activin type 2 receptor gene (44.4%), DKFZp564K112 (a homologue of the Drosophila tumor suppressor gene multi-sex-combs; 41.2%), and an endoplasmic reticulum chaperone protein gene SEC63 (37.5%). The mutational spectra for genes with high mutation frequencies were also significantly different between MSI-H gastric and colorectal cancers.

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عنوان ژورنال:
  • Cancer research

دوره 62 13  شماره 

صفحات  -

تاریخ انتشار 2002